subject: Online Cpr - Omega-3 And Sudden Cardiac Arrest [print this page] Sudden Cardiac Death (SCD) is accountable for a lot of deaths within the U.S. and other countries around the world. It's not the exact same as a heart attack. A heart attack commonly occurs when progressed atherosclerosis (blocking of the arteries) gradually starves the heart causing irreparable damage. The heart finally can't operate properly and stops. Sudden cardiac death happens when the electrical impulses that manage heart function turn out to be erratic resulting in an irregular heartbeat (arrhythmia). When the arrhythmia is severe enough the heart abruptly stops, starving the brain of needed blood. Death often follows rapidly unless emergency care is given instantly. Frequently sudden cardiac death occurs when no other heart illness is detected.
It has long been recognized that ingesting fish can reduce the likelihood of heart attack along with other cardiovascular diseases. The reason? Fish, particularly cold-water fatty fish, is high in two omega-3 polyunsaturated fatty acids (PUFA). These two fatty acids are eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA). Omega-3 has been shown to decrease the build-up of artery clogging atherosclerosis and keep blood platelets from sticking collectively therefore lowering the risk of heart attack and stroke.
But more recent research suggest that omega-3 fatty acids also have an impact on the threatening arrhythmias that may cause sudden cardiac death by managing the impulses that control heart rhythm. 1 study included the use of mycocytes (cells that beat independently). By incorporating the totally free fatty acids to these cells arrhythmias were aborted. When the fatty acids were taken out from the mycocytes the arrhythmias would reoccur therefore implying that omega-3 fatty acids have a stabilizing effect on center rhythm. The apparent system for managing rhythm involved cell ion channels, proteins that control the movement of sodium, calcium and potassium ions through the membrane from the cell.
Another study, executed by Danish scientists, examined the relationship between center rate variability and omega-3 fatty acids in wholesome subjects. It had already been established that heart arrhythmias could be absolutely impacted in patients who had been recent heart attack victims. These researchers wanted to determine if people otherwise totally free of heart disease could gain the same arrhythmia preventing advantages from omega-3 fatty acids.
Sixty healthy adults had been arbitrarily split into three groups. The first group got day-to-day supplements containing 6.6 g of omega-3 (containing 3.0g EPA and 2.9g DHA). Group two received 2.0g of omega-3 (containing 0.9g EPA and 0.8g DHA). The remaining group received only an olive oil placebo. The supplements were given for 12 weeks. Before and after the supplements had been consumed heart rate variability and blood cell fatty acids had been measured. It was discovered that heart rate variability was favorably influenced by the amount of omega-3 consumed. In other words, the much more omega-3 1 consumed the higher the heart rate variability, particularly in men with low heart rate variability prior to supplementing.
The researchers concluded that omega-3 fatty acids positively affected heart rate variability thus having a protective effect on heart function. These finding were similar to those of earlier studies showing that omega-3 positively affects heart rhythm in patients who had suffered a previous heart attack. Since sudden cardiac death is the result of erratic heart rhythm, omega-3's heart protective qualities show great promise in the fight against sudden cardiac arrest. The mechanical studies completed by these along with other researchers seemed to confirm other studies that the oral ingestion of fish and fish oils supply prophylaxis for the prevention of fatal cardiac arrhythmias when taken regularly in small amounts. In simple terms, consuming fish and fish oil supplements can save lives.
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